A CNIC-led study identifies a mitochondrial “checkpoint” that enables dendritic cells to efficiently activate T lymphocytes against viruses and tumors
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A new CNIC-led study shows that a genetic tool derived from baker’s yeast enables human cells to manufacture the building blocks of DNA even when their mitochondria fail.
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Using gene editing with CRISPR-Cas9, the team has been able to study these proteins without altering them or producing them in excess, thereby allowing observation of their true function.
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The presence of this protein slows down the activity of the mitochondria, decreasing their ability to eliminate toxic substances such as certain bile acids.
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CNIC researchers have defined the dynamics of the transfer of mitochondrial DNA from mothers to their offspring
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Researchers at the Centre for Genomic Regulation shed light on a crucial moment in the evolution of life: when cells acquired mitochondria.
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